16. SYSTEM CONTROLS/COMPLEMENT REGULATION

Yves Laure Pimentel · 70問 · 2年前

通報

問題一覧

Complement if not controlled may lead to

HEALTHY tissue destruction and autoimmune disease

Dissociates C1r and C1s from C1q and inactivates MASP-2

C1 Inhibitor (C1INH)

Factor I function

Cleaves C3b and C4b

Factor I

, found on PBS, has the greatest affinity to C3b

CR1 (comp. receptor type 1) -or CD35

,Found on all epithelial and endothelial cells except RBC, most EFFICIENT cofactor for Factor I.

MCP (membrane Cofactor Protein) or CD46

MCP (membrane Cofactor Protein) or

CD46

or CD46

MCP (membrane Cofactor Protein)

CR1 (comp. receptor type 1) -or

CD35

-or CD35

CR1 (comp. receptor type 1)

DAF (decay accelerating factor) - or

CD55

or CD55

DAF (decay accelerating factor) -

found on endothelial cells, fibroblast, PBS and epithelial cells. DISSOCIATE C3 convertase

DAF (decay accelerating factor) - or CD55,

its presence protects host cells from by STANDER lysis

DAF (decay accelerating factor) - or CD55,

prevents binding of B to C3b (alternative)

Factor H

Factor H prevents binding of ? (alternative)

B to C3b

Acts as a cofactor with Factor I to inactivate C4b (C4bp)

C4b binding protein

Prevents attachment of the C5b67 complex to cell membranes

S protein (vitronectin)

S protein (?)

vitronectin

? (vitronectin)

S protein

widely distributed on all cells. It’s main function is to bind to C8 and prevent insertion of C9 into host cell

BOUND RECEPTOR MIRL (Membrane Inhibitor Reactive Lysis) - Or CD59,

other names parasitology

other names parasitology

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CSF 2

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SEMEN 1

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SEMEN 2

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SYNOVIAL FLUID 1

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SYNOVIAL FLUID 2

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SEROUS FLUID

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PLEURAL FLUID

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PERICARDIAL FLUID

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AMNIOTIC FLUID

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FECALYSIS

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