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Psychopharmacology Exam 2 (part 1)

Psychopharmacology Exam 2 (part 1)
19問 • 1年前
  • Two Clean Queens
  • 通報

    問題一覧

  • 1

    Benefits: Unknown SE: Weight Gain

    5-HT2C

  • 2

    Benefits: Sedation SE: Sedation, increased appetite, weight gain, hypotension

    H1

  • 3

    Benefits: Low EPS SE: Autonomic SE such as blurred vision, dry mouth, constipation, urinary retention, tachycardia; memory dysfunction

    M1

  • 4

    Has high H1 and M1, increased sedation and anticholinergic effects, but lower EPS effects.

    Low potency (eg Chlorpromazine)

  • 5

    Has low H1 and M1 receptor activity. Has little sedation, weight gain, or anticholinergic activity. High risk for EPS effects.

    High potency (eg Haldol)

  • 6

    Select the high potency 1st Gen Antipsychotic

    Droperidol

  • 7

    Select the high potency 1st Gen Antipsychotic

    Haloperidol

  • 8

    Select the high potency 1st Gen Antipsychotic

    Loxapine

  • 9

    Select the high potency 1st Gen Antipsychotic

    Pimozide

  • 10

    Select the high potency 1st Gen Antipsychotic

    Thiothixene

  • 11

    Select the high potency 1st Gen Antipsychotic

    Trifluoperazine

  • 12

    Receptors Blocked: D2 (moderate), Acetylcholine M1 (strong), H1 (strong), Alpha (moderate), and D1, D3, D4, D5 (variable)

    Low potency 1st Gen Antipsychotics (eg. Chlorpromazine)

  • 13

    Receptors Blocked: D2 (strong), Acetylcholine M1 (weak), H1 (weak), Alpha (weak), and D1, D3, D4, D5 (variable)

    High potency (eg Haloperidol)

  • 14

    Select the correct symptoms on NMS

    Onset: Days to weeks NM findings: Bradyreflexia, severe muscle rigidity (lead pipe) Causative agents: Dopamine antagonists Treatments: Bromocriptine Resolution: Days to weeks

  • 15

    Select the symptoms of SS

    Onset: Within 24 hours NM findings: Hyperreflexia (tremor, clonus) Causative agents: Serotonin agonist Treatments: Benzodiazepine, cyproheptadine Resolution: Within 24 hrs

  • 16

    Other than Bromocriptine, what other agents could you give to treat NMS in addition to supportive care?

    IV Benzos, IV Dantrolene (mm relaxant), and Amantadine (dopamine agonist)

  • 17

    Low potency D2 antagonist that antagonizes many receptors, the only 1st Gen to lower seizure risk, can cause photosensitivity (blue/gray skin) and corneal deposits in eyes.

    Chlorpromazine

  • 18

    High potency D2 antagonist, lacks anticholinergic activity (used often for delirium), but high risk of EPS/TD and prolonged QTc (IV route)

    Haloperidol

  • 19

    Explain differences in treatment management of restarting D2 antagonsits after Serotonin Syndrome vs NMS?

    Restarting D2 antagonsits after Serotonin Syndrome is prohibited, but you can restart them 2 weeks after NMS has resolved keeping in mind to avoid use of high-potency D2 antagonsits, lithium, and dehydration.

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    Psychopathology

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    問題一覧

  • 1

    Benefits: Unknown SE: Weight Gain

    5-HT2C

  • 2

    Benefits: Sedation SE: Sedation, increased appetite, weight gain, hypotension

    H1

  • 3

    Benefits: Low EPS SE: Autonomic SE such as blurred vision, dry mouth, constipation, urinary retention, tachycardia; memory dysfunction

    M1

  • 4

    Has high H1 and M1, increased sedation and anticholinergic effects, but lower EPS effects.

    Low potency (eg Chlorpromazine)

  • 5

    Has low H1 and M1 receptor activity. Has little sedation, weight gain, or anticholinergic activity. High risk for EPS effects.

    High potency (eg Haldol)

  • 6

    Select the high potency 1st Gen Antipsychotic

    Droperidol

  • 7

    Select the high potency 1st Gen Antipsychotic

    Haloperidol

  • 8

    Select the high potency 1st Gen Antipsychotic

    Loxapine

  • 9

    Select the high potency 1st Gen Antipsychotic

    Pimozide

  • 10

    Select the high potency 1st Gen Antipsychotic

    Thiothixene

  • 11

    Select the high potency 1st Gen Antipsychotic

    Trifluoperazine

  • 12

    Receptors Blocked: D2 (moderate), Acetylcholine M1 (strong), H1 (strong), Alpha (moderate), and D1, D3, D4, D5 (variable)

    Low potency 1st Gen Antipsychotics (eg. Chlorpromazine)

  • 13

    Receptors Blocked: D2 (strong), Acetylcholine M1 (weak), H1 (weak), Alpha (weak), and D1, D3, D4, D5 (variable)

    High potency (eg Haloperidol)

  • 14

    Select the correct symptoms on NMS

    Onset: Days to weeks NM findings: Bradyreflexia, severe muscle rigidity (lead pipe) Causative agents: Dopamine antagonists Treatments: Bromocriptine Resolution: Days to weeks

  • 15

    Select the symptoms of SS

    Onset: Within 24 hours NM findings: Hyperreflexia (tremor, clonus) Causative agents: Serotonin agonist Treatments: Benzodiazepine, cyproheptadine Resolution: Within 24 hrs

  • 16

    Other than Bromocriptine, what other agents could you give to treat NMS in addition to supportive care?

    IV Benzos, IV Dantrolene (mm relaxant), and Amantadine (dopamine agonist)

  • 17

    Low potency D2 antagonist that antagonizes many receptors, the only 1st Gen to lower seizure risk, can cause photosensitivity (blue/gray skin) and corneal deposits in eyes.

    Chlorpromazine

  • 18

    High potency D2 antagonist, lacks anticholinergic activity (used often for delirium), but high risk of EPS/TD and prolonged QTc (IV route)

    Haloperidol

  • 19

    Explain differences in treatment management of restarting D2 antagonsits after Serotonin Syndrome vs NMS?

    Restarting D2 antagonsits after Serotonin Syndrome is prohibited, but you can restart them 2 weeks after NMS has resolved keeping in mind to avoid use of high-potency D2 antagonsits, lithium, and dehydration.