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Susceptible host
  • Tameyra Stefani Al-Suhairy

  • 問題数 73 • 11/9/2024

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    問題一覧

  • 1

    Substantially reduce sucrose from diet Eliminate sucrose from between meal snacks

    Limit substrate

  • 2

    due to high salivary

    Congenital chloride diarrhea

  • 3

    Gross carious teeth are seen

    Rapp-Hodgkin ectodermal dysplasia

  • 4

    Varied appearance, ranging from a shiny, white, opaque or discolored spot in the enamel to a hard, dark dentinal surface exposed to the oral environment

    Arrested lesion

  • 5

    - commonly without clinical symptoms - hard, dry, and dark brown - very similar clinical appearance as arrested caries lesions

    Slowly progressing lesions

  • 6

    Excessive fluoride exposure (10 PPM/ more) results in

    Fluorosis

  • 7

    substances is facilitated by the flushing action of saliva

    Oral clearance

  • 8

    TRANSPARENT DENTIN Zone of carious dentin that is softer than normal dentin. Shows further loss of mineral from intertubular dentin and many large crystals in the lower of dentinal tubules

    Zone 3

  • 9

    Clinically observed as intact, but discolored usually brown or black spots.

    Arrested Caries ( mineralized lesion)

  • 10

    Lesions found adjacent to restorations

    Secondary caries lesions (Recurrent caries)

  • 11

    Due to inter-dental spacing.

    Turner syndrome

  • 12

    • It recognized clinically, as a wet, mushy, easily removable mass. • This is structure less/ granular in histologic appearance and contain masses of bacteria.

    Necrotic dentin

  • 13

    Zones 3 and 4 - not significantly discolored - feels hard already - capable of remineralization

    Affected dentin

  • 14

    Often painful or hypersensitive - soft, wet and light yellow - demineralization penetrates deep into dentin

    Rapidly progressing lesions

  • 15

    In 17 th and 18 th century,there emerged a concept that teeth are destroyed by acids formed in the oral cavity by fermentation of food particles around the teeth

    Chemical Theory

  • 16

    TURBID DENTIN Zones of bacterial invasion marked by widening and distortion of dentinal tubules w/c filled with bacteria. Dentin in this zone will not self repair.

    Zone 4

  • 17

    Fairly hard or leathery texture and highly discolored

    Chronic (Slowly progressing) Caries

  • 18

    least resistance to caries attach and allows rapid lateral spreading once caries has penetrated the enamel.

    DEJ

  • 19

    Proteolytic theory

  • 20

    Lubrication

    mucins

  • 21

    due to delayed or retarded eruption with consequently increased time for enamel maturation before exposure to the oral environment

    Growth hormone deficiency

  • 22

    3 Smooth enamel surfaces that shelter plaque

    areas cervical to the contact areas, distal surface of most posterior tooth, areas cervical to the heights of contour on the facial and lingual

  • 23

    was the first well known scientist and investigator of dental caries and published his results in 1882.

    WD miller

  • 24

    5 BUFFERING SYSTEMS

    Bicarbonate, phosphate, proteins, sialin, urea

  • 25

    (a proline-rich peptide) stabilizes Ca and PO4 ions and prevents excessive deposition of the ions to teeth

    Statherin

  • 26

    change in color is presumambly due to trapped organic debris and mettalic ions with the enamel.

    Arrested Caries ( mineralized lesion)

  • 27

    due to aversion to sweet foods

    Hereditary fructose intolerance

  • 28

    process of neutralizing any added acid (H+ ions) or base (OH- ions) to maintain the moderate pH, making them a weaker acid or base.

    Buffering

  • 29

    Hard, shiny, smooth surface with brown- black discoloration

    Arrested Caries

  • 30

    areas are protected physically and are relatively free from the effects of mastication, tongue movement, and salivary flow.

    Smooth enamel surfaces

  • 31

    populated by organism with receptors specialized for attachment to the surface of epithelium.

    Oral Mucosa

  • 32

    plaque at the dorsum of the tongue.

    Streptococcus Salivarius

  • 33

    Possibly related to delayed eruption & inter-dental spacing.

    Down syndrome

  • 34

    gross carious tooth is one of the clinical features

    Focal dermal hypoplasia:

  • 35

    normal daily secretion of saliva

    0.5 - 1.5 liters.

  • 36

    alteration in the enamel and an increased caries incidence.

    Epidermolysis bullosa

  • 37

    Type of caries according to extent 2

    Incipient or reversible, Cavitated or irreversible

  • 38

    Digestion

    amylase and lipase

  • 39

    INFECTED DENTIN Outermost zone of infected dentin Decomposed dentin that is teeming with bacteria. No recognizable structure to dentin, seems to be absent of collagen and mineral.

    Zone 5

  • 40

    Morphologic and Structural Characteristics of Teeth that Increase Tooth Susceptibility to Dental caries

    Aberrant anatomic and morphologic configurations, Rough tooth surface, Tooth surfaces that are non-self cleansing

  • 41

    5 Antimicrobial

    Ig, peroxidases, lysozyme, lactoferrins, histatins

  • 42

    due to prolonged antibiotic therapy

    Primary immunodeficiency

  • 43

    produces abundant quantities of watery saliva

    Parasympathetic stimulation

  • 44

    mineralization occurs after cavitations , the remaining exposed surface becomes harder and softer and often becomes dark brown/ black in color

    Arrested caries

  • 45

    Factors needed cares initiation 4

    Flora bacteria in plaque, Diet, Susceptible surface, Time

  • 46

    produces more viscous saliva.

    sympathetic stimulatio

  • 47

    whole unstimulated saliva flow rate is approximately

    0.3-0.4 ml / min

  • 48

    Proteolytic theory

  • 49

    Goal of diet counseling is to identify source of sucrose in the diet and reduce the intake of these foods

    Limit substrate

  • 50

    also known as dry mouth,is dryness in the mouth, which may be associated with a change in the composition of saliva, or reduced salivary flow.

    Xerostomia

  • 51

    Microorganisms were associated with the carious process

    Parasitic theory

  • 52

    present in the oral cavity of almost all individuals.

    Mutans streptococci (MS)

  • 53

    The marked caries results from poor dental care resulting from a small mouth opening, misalignment of the teeth & mental retardation

    Rubinstein-Taybi syndrome

  • 54

    Bactericidal mouth rinses (chlorhexidine) • Topical fluoride treatments • Antibiotic treatment (vancomycin, tetracycline, polymyxin B)

    Modify Microflora

  • 55

    Acidogenic theory

  • 56

    3 minerals

    Calcium, Phosphate, Fluoride

  • 57

    Zones 1 and 2 - significantly discolored - can be removed by excavators - stained with caries detector - needs to be removed unless judged to be within 0.5 mm of pulp

    Infected dentin

  • 58

    Soft, light- colored lesion

    Acute (Rampant) Caries

  • 59

    Importance of Saliva in the maintenance of normal flora (4)

    Bacterial clearance, Direct Antibacterial Activity, Buffer, Demineralization

  • 60

    SUB TRANSPARENT DENTIN Zone of demineralized (by acid from caries) of the intertubular dentin and initial formation of very fine crystals in the tubules and lumen at the advancing prone. Damage to the odontoblastic process is ended, however no bacteria are founf in this zone.

    Zone 2

  • 61

    NORMAL DENTIN Has a tubule with odontoblastics rocesses that are smooth and no crystals in the line. no bacteria in the tubules

    Zone 1

  • 62

    Enhances post-eruption maturation of teeth Remineralizes demineralized tooth structures

    Minerals

  • 63

    dominated the teeth normally have plaque community.

    Sanguis and S.Mitis

  • 64

    Lesions found in intact tooth surfaces

    Primary caries lesions

  • 65

    Rationale: Eliminate MS from mouth

    Modify Microflora

  • 66

    Rationale: Reduce number, duration and intensity of acid attacks. Reduce selection pressure for MS

    Limit substrate

  • 67

    Base of pits and fissures

  • 68

    Increased susceptibility to caries has been found

    Klinefelter syndrome

  • 69

    due to high salivary pH

    Chronic renal failure

  • 70

    brownish discoloration of enamel term

    Mottled enamel

  • 71

    Health History Factors Associated with Increased Caries Risk 6

    Age, Fluoride exposure, Smoking, Alcohol, General health, Medication

  • 72

    In enamel, it has a dull, white, opaque appearance. In dentin, a soft, yellowish or light to dark brown discolorations of the demineralized tissues prevail.

    Active lesion

  • 73

    Caries lesion left behind in the preparation or at the enamel margin of a preparation when a restoration is placed

    Remaining caries lesions