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1
Substantially reduce sucrose from diet Eliminate sucrose from between meal snacks
Limit substrate
2
due to high salivary
Congenital chloride diarrhea
3
Gross carious teeth are seen
Rapp-Hodgkin ectodermal dysplasia
4
Varied appearance, ranging from a shiny, white, opaque or discolored spot in the enamel to a hard, dark dentinal surface exposed to the oral environment
Arrested lesion
5
- commonly without clinical symptoms - hard, dry, and dark brown - very similar clinical appearance as arrested caries lesions
Slowly progressing lesions
6
Excessive fluoride exposure (10 PPM/ more) results in
Fluorosis
7
substances is facilitated by the flushing action of saliva
Oral clearance
8
TRANSPARENT DENTIN Zone of carious dentin that is softer than normal dentin. Shows further loss of mineral from intertubular dentin and many large crystals in the lower of dentinal tubules
Zone 3
9
Clinically observed as intact, but discolored usually brown or black spots.
Arrested Caries ( mineralized lesion)
10
Lesions found adjacent to restorations
Secondary caries lesions (Recurrent caries)
11
Due to inter-dental spacing.
Turner syndrome
12
• It recognized clinically, as a wet, mushy, easily removable mass. • This is structure less/ granular in histologic appearance and contain masses of bacteria.
Necrotic dentin
13
Zones 3 and 4 - not significantly discolored - feels hard already - capable of remineralization
Affected dentin
14
Often painful or hypersensitive - soft, wet and light yellow - demineralization penetrates deep into dentin
Rapidly progressing lesions
15
In 17 th and 18 th century,there emerged a concept that teeth are destroyed by acids formed in the oral cavity by fermentation of food particles around the teeth
Chemical Theory
16
TURBID DENTIN Zones of bacterial invasion marked by widening and distortion of dentinal tubules w/c filled with bacteria. Dentin in this zone will not self repair.
Zone 4
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Fairly hard or leathery texture and highly discolored
Chronic (Slowly progressing) Caries
18
least resistance to caries attach and allows rapid lateral spreading once caries has penetrated the enamel.
DEJ
19
Proteolytic theory
20
Lubrication
mucins
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due to delayed or retarded eruption with consequently increased time for enamel maturation before exposure to the oral environment
Growth hormone deficiency
22
3 Smooth enamel surfaces that shelter plaque
areas cervical to the contact areas, distal surface of most posterior tooth, areas cervical to the heights of contour on the facial and lingual
23
was the first well known scientist and investigator of dental caries and published his results in 1882.
WD miller
24
5 BUFFERING SYSTEMS
Bicarbonate, phosphate, proteins, sialin, urea
25
(a proline-rich peptide) stabilizes Ca and PO4 ions and prevents excessive deposition of the ions to teeth
Statherin
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change in color is presumambly due to trapped organic debris and mettalic ions with the enamel.
Arrested Caries ( mineralized lesion)
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due to aversion to sweet foods
Hereditary fructose intolerance
28
process of neutralizing any added acid (H+ ions) or base (OH- ions) to maintain the moderate pH, making them a weaker acid or base.
Buffering
29
Hard, shiny, smooth surface with brown- black discoloration
Arrested Caries
30
areas are protected physically and are relatively free from the effects of mastication, tongue movement, and salivary flow.
Smooth enamel surfaces
31
populated by organism with receptors specialized for attachment to the surface of epithelium.
Oral Mucosa
32
plaque at the dorsum of the tongue.
Streptococcus Salivarius
33
Possibly related to delayed eruption & inter-dental spacing.
Down syndrome
34
gross carious tooth is one of the clinical features
Focal dermal hypoplasia:
35
normal daily secretion of saliva
0.5 - 1.5 liters.
36
alteration in the enamel and an increased caries incidence.
Epidermolysis bullosa
37
Type of caries according to extent 2
Incipient or reversible, Cavitated or irreversible
38
Digestion
amylase and lipase
39
INFECTED DENTIN Outermost zone of infected dentin Decomposed dentin that is teeming with bacteria. No recognizable structure to dentin, seems to be absent of collagen and mineral.
Zone 5
40
Morphologic and Structural Characteristics of Teeth that Increase Tooth Susceptibility to Dental caries
Aberrant anatomic and morphologic configurations, Rough tooth surface, Tooth surfaces that are non-self cleansing
41
5 Antimicrobial
Ig, peroxidases, lysozyme, lactoferrins, histatins
42
due to prolonged antibiotic therapy
Primary immunodeficiency
43
produces abundant quantities of watery saliva
Parasympathetic stimulation
44
mineralization occurs after cavitations , the remaining exposed surface becomes harder and softer and often becomes dark brown/ black in color
Arrested caries
45
Factors needed cares initiation 4
Flora bacteria in plaque, Diet, Susceptible surface, Time
46
produces more viscous saliva.
sympathetic stimulatio
47
whole unstimulated saliva flow rate is approximately
0.3-0.4 ml / min
48
Proteolytic theory
49
Goal of diet counseling is to identify source of sucrose in the diet and reduce the intake of these foods
Limit substrate
50
also known as dry mouth,is dryness in the mouth, which may be associated with a change in the composition of saliva, or reduced salivary flow.
Xerostomia
51
Microorganisms were associated with the carious process
Parasitic theory
52
present in the oral cavity of almost all individuals.
Mutans streptococci (MS)
53
The marked caries results from poor dental care resulting from a small mouth opening, misalignment of the teeth & mental retardation
Rubinstein-Taybi syndrome
54
Bactericidal mouth rinses (chlorhexidine) • Topical fluoride treatments • Antibiotic treatment (vancomycin, tetracycline, polymyxin B)
Modify Microflora
55
Acidogenic theory
56
3 minerals
Calcium, Phosphate, Fluoride
57
Zones 1 and 2 - significantly discolored - can be removed by excavators - stained with caries detector - needs to be removed unless judged to be within 0.5 mm of pulp
Infected dentin
58
Soft, light- colored lesion
Acute (Rampant) Caries
59
Importance of Saliva in the maintenance of normal flora (4)
Bacterial clearance, Direct Antibacterial Activity, Buffer, Demineralization
60
SUB TRANSPARENT DENTIN Zone of demineralized (by acid from caries) of the intertubular dentin and initial formation of very fine crystals in the tubules and lumen at the advancing prone. Damage to the odontoblastic process is ended, however no bacteria are founf in this zone.
Zone 2
61
NORMAL DENTIN Has a tubule with odontoblastics rocesses that are smooth and no crystals in the line. no bacteria in the tubules
Zone 1
62
Enhances post-eruption maturation of teeth Remineralizes demineralized tooth structures
Minerals
63
dominated the teeth normally have plaque community.
Sanguis and S.Mitis
64
Lesions found in intact tooth surfaces
Primary caries lesions
65
Rationale: Eliminate MS from mouth
Modify Microflora
66
Rationale: Reduce number, duration and intensity of acid attacks. Reduce selection pressure for MS
Limit substrate
67
Base of pits and fissures
68
Increased susceptibility to caries has been found
Klinefelter syndrome
69
due to high salivary pH
Chronic renal failure
70
brownish discoloration of enamel term
Mottled enamel
71
Health History Factors Associated with Increased Caries Risk 6
Age, Fluoride exposure, Smoking, Alcohol, General health, Medication
72
In enamel, it has a dull, white, opaque appearance. In dentin, a soft, yellowish or light to dark brown discolorations of the demineralized tissues prevail.
Active lesion
73
Caries lesion left behind in the preparation or at the enamel margin of a preparation when a restoration is placed
Remaining caries lesions