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問題一覧
1
Irreversible cell injury (necrosis) light microscope
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Metaplasia
3
Atrophy mechanism
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Fibrinoid necrosis
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Hyperplasia, hypertrophy
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Mechanism:
Atrophy
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Acquired
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Cellular aging
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Fat necrosis
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Symptom
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Inflammation necrosis
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Small spindle shaped uterine Smooth muscle cells (normal uterus)
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Atrophy
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Hormonal physiologic hyperplasia
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Physiologic atrophy
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Pathogenesis
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Coagulative necrosis
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Pyknosis
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Connective tissue metaplasia
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Karyorrhexis
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Morphologic changes
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Unknown cause of a disease Cryptogenic, essential, spontaneous
Idiopathic disease
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Reversible cell injury (light microscope)
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Large plump cells (gravid uterus)
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Compensatory physiologic hyperplasia
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Excess growth factors pathologic hyperplasia
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Atrophy
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Reversible
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hypertrophy physiologic
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Coagulative necrosis
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Basic reactions of cell Tissues to abnormal stimuli Inherited defect that underlie all disease
General pathology
32
Mechanism:
Atrophy
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Hypertrophy uterus during pregnancy
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Clinical manifestation
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Metaplasia
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Caseous necrosis (cheeselike)
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5 patterns tissue necrosis
Coagulative, Liquefactive, Caseous, Fat, Fibrinoid
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Hypertrophy
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Karyolysis
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Irreversible cell injury (necrosis)
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Atrophy
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4 aspects of disease process forming the core of pathology
Etiology, Pathogenesis, Morphologic changes, Clinical manifestations
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Genetic
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Pathologic atrophy
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Pathologic hypertrophy
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mechanism
Metaplasia
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Studies specific responses of specialized organs and tissues Responsible for disorders
Special or systemic pathology
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Metaplasia
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Metaplasia
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Hyperplasia
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Signs
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Metaplasia
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Cellular adaptations
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Excess hormones pathologic hyperplasia
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Irreversible
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Normal
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Mechanism:
Metaplasia
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Liquefactive necrosis
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Mechanism:
Metaplasia
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Multifactorial
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Hyperplasia
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Hypertrophy