Histopath (somatic death)

25問 • 2年前

Kmyching

通報

問題一覧

PROGRAM CELL DEATH”. vital process that helps eliminate unwanted cells

apoptosis

physiologic death of cells

necrobiosis

consists of more or less rapid coagulation cytoplasm. Encountered when there is infarction. most common “TOMBSTONE FORMATION”

Coagulation necrosis

due to enzymes, necrosis of tissue rich in liquid. brain and spinal cord

Liquefaction necrosis

yellow, cheesy, crumbly material

Caseous necrosis

SULFIDE GAS FORMATION”

gangrenous necrosis

arterial occlusion

Dry gangrene

Venous occlusion

Wet gangrene

involves the destruction of adipose tissue. Fat destruction due to release of pancreatic lipases. “CHALKY WHITE APPEARANCE”

Fat necrosis

indicates reduction in size and condensation of the nuclear material

Pyknosis

indicates the segmentation and fragmentation of the nucleus, whereby nuclear contents are broken up and released into the cytoplasm

Karyorrhexis

means the dissolution of the nucleus where all basophilism is lost and the nucleus disappears

Karyolysis

The cell may appear larger SWELLING’’

Cytoplasmic changes

Refers to the death or complete cessation of metabolic and functional activities of the organism or body Cells survive 4-6 minutes after lungs and heart stop functioning

Somatic death

death because of cardiovascular integrity

Circulatory failure

leads to death due to absence of oxygen and accumulation of carbon dioxide

Respiratory failure

causing loss of coordination of various body functions

Nervous failure (CNS failure)

this is the first demonstrable change observed, characterized by cooling of the body. 7°F PER HOUR

ALGOR MORTIS

refers to the rigidity or stiffening of the muscles, occurring 6-12hours after death.

RIGOR MORTIS

refers to the purplish discoloration or lividity of the skin. Due to stasis and settling down of blood.

LIVOR MORTIS

refers to the drying and wrinkling of the cornea and the anterior chamber of the eye

DESSICATION

characterized by foul-smelling gases, due to multiplying saprophytic organisms

PUTREFACTION

this implies self-digestion of the cells, bacteria enhance the destruction of tissue.

AUTOLYSIS

pathologic death of cell which is due to disease or injury.

Necrosis

yellow, cheesy, crumbly material

Caseous necrosis

HISTOPATH (lesson 1)

Kmyching · 56問 · 2年前

HISTOPATH (lesson 1)

56問 • 2年前

Kmyching

bacte (microbial control)

Kmyching · 26問 · 2年前

bacte (microbial control)

Collection

Collection

fresh tissue

fresh tissue

Staphylococcus

Staphylococcus

lesson 1 (cell injury, adaptation and inflammation and repair)

Kmyching · 67問 · 2年前

lesson 1 (cell injury, adaptation and inflammation and repair)

biopsy

biopsy

autopsy

autopsy

decalcification

decalcification

DEHYDRATION AND CLEARING

Kmyching · 27問 · 2年前

DEHYDRATION AND CLEARING

27問 • 2年前

Kmyching

IMPREGNATION AND EMBEDDING

Kmyching · 21問 · 2年前

IMPREGNATION AND EMBEDDING

21問 • 2年前

Kmyching

TRIMMING AND SECTIONING

Kmyching · 35問 · 2年前

TRIMMING AND SECTIONING

35問 • 2年前

Kmyching

adhesive and staining

Kmyching · 43問 · 2年前

adhesive and staining

43問 • 2年前

Kmyching

COMMON STAINING SOLUTION

Kmyching · 19問 · 2年前

COMMON STAINING SOLUTION

OTHER STAINING

OTHER STAINING

H&E STAINING, MOUNTING, RINGING AND LABELLING

Kmyching · 19問 · 1年前

H&E STAINING, MOUNTING, RINGING AND LABELLING

IMMUNOHISTOCHEMISTRY

IMMUNOHISTOCHEMISTRY

EPITHELIAL TUMOR MARKERS

Kmyching · 5問 · 1年前

EPITHELIAL TUMOR MARKERS

5問 • 1年前

Kmyching

INTERMEDIATE FILAMENT MARKERS

Kmyching · 6問 · 1年前

INTERMEDIATE FILAMENT MARKERS

6問 • 1年前

Kmyching

NEUROENDOCRINE MARKERS and GERM CELL TUMOR MARKERS

Kmyching · 6問 · 1年前

NEUROENDOCRINE MARKERS and GERM CELL TUMOR MARKERS

6問 • 1年前

Kmyching

MESENCHYMAL TUMOR MARKERS

Kmyching · 5問 · 1年前

MESENCHYMAL TUMOR MARKERS

5問 • 1年前

Kmyching

問題一覧

PROGRAM CELL DEATH”. vital process that helps eliminate unwanted cells

apoptosis

physiologic death of cells

necrobiosis

consists of more or less rapid coagulation cytoplasm. Encountered when there is infarction. most common “TOMBSTONE FORMATION”

Coagulation necrosis

due to enzymes, necrosis of tissue rich in liquid. brain and spinal cord

Liquefaction necrosis

yellow, cheesy, crumbly material

Caseous necrosis

SULFIDE GAS FORMATION”

gangrenous necrosis

arterial occlusion

Dry gangrene

Venous occlusion

Wet gangrene

involves the destruction of adipose tissue. Fat destruction due to release of pancreatic lipases. “CHALKY WHITE APPEARANCE”

Fat necrosis

indicates reduction in size and condensation of the nuclear material

Pyknosis

indicates the segmentation and fragmentation of the nucleus, whereby nuclear contents are broken up and released into the cytoplasm

Karyorrhexis

means the dissolution of the nucleus where all basophilism is lost and the nucleus disappears

Karyolysis

The cell may appear larger SWELLING’’

Cytoplasmic changes

Refers to the death or complete cessation of metabolic and functional activities of the organism or body Cells survive 4-6 minutes after lungs and heart stop functioning

Somatic death

death because of cardiovascular integrity

Circulatory failure

leads to death due to absence of oxygen and accumulation of carbon dioxide

Respiratory failure

causing loss of coordination of various body functions

Nervous failure (CNS failure)

this is the first demonstrable change observed, characterized by cooling of the body. 7°F PER HOUR

ALGOR MORTIS

refers to the rigidity or stiffening of the muscles, occurring 6-12hours after death.

RIGOR MORTIS

refers to the purplish discoloration or lividity of the skin. Due to stasis and settling down of blood.

LIVOR MORTIS

refers to the drying and wrinkling of the cornea and the anterior chamber of the eye

DESSICATION

characterized by foul-smelling gases, due to multiplying saprophytic organisms

PUTREFACTION

this implies self-digestion of the cells, bacteria enhance the destruction of tissue.

AUTOLYSIS

pathologic death of cell which is due to disease or injury.

Necrosis

yellow, cheesy, crumbly material

Caseous necrosis